Clonal stem‑cell disorders causing excess proliferation of one or more myeloid lineages (RBCs, WBCs, platelets). Often associated with JAK2 V617F mutation (except CML → BCR‑ABL)

Disorder Main Cell Line Hallmark Findings Key Mutation Notes
Polycythaemia vera (PV) RBCs ↑ Hb/Hct, pruritus after hot bath, plethora, splenomegaly JAK2+ Erythropoietin ↓
Essential thrombocythaemia (ET) Platelets Persistent thrombocytosis (>450 ×10⁹/L), microvascular symptoms (erythromelalgia, headache), splenomegaly JAK2+ (50–60%), CALR/MPL Risk of thrombosis & bleeding
Primary myelofibrosis (PMF) Fibroblast activation → marrow fibrosis Leukoerythroblastic blood film, tear‑drop RBCs, massive splenomegaly JAK2+ (50%), CALR/MPL “Dry tap” on marrow aspirate
Chronic myeloid leukaemia (CML) Granulocytes Marked leukocytosis, basophilia, splenomegaly BCR‑ABL (Philadelphia chromosome) Responds to imatinib

Management Overview

Disorder First‑line Additional
PV Venesection ± hydroxycarbamide Aspirin for thrombosis prevention
ET Hydroxycarbamide (high‑risk) Aspirin (low‑risk)
PMF Supportive, ruxolitinib (JAK inhibitor) Stem‑cell transplant (young fit)
CML Imatinib (tyrosine kinase inhibitor) Alternatives: dasatinib, nilotinib

Myeloproliferative neoplasms (MPN)

The most clinically relevant MPN include chronic myeloid leukemia (CML), polycythemia vera (PV), primary myelofibrosis (PMF), and essential thrombocythemia (ET).

Chronic eosinophilic leukemia is also a type of MPN.

All myeloproliferative neoplasms may lead to elevated uric acid levels and gout as a result of increased cellular breakdown. They are also associated with an increased risk of acute myeloid leukemia.

Chronic Myelogenous Leukemia (CML)

Thrombocytosis

Polycythemia Vera

Agnogenic Myeloid Metaplasia

Tx

Allogenic hematopoietic cell transplantation (allo-HCT) for patients under 60 yo age

Palliative treatment - Median survival time is 13 months for patients who are