Post-MI Complications
Patients are at risk of a number of immediate, early and late complications following a myocardial infarction (MI).
This most commonly occurs due to patients developing ventricular fibrillation and is the most common cause of death following a MI. Patients are managed as per the ALS protocol with defibrillation.
If a large part of the ventricular myocardium is damaged in the infarction the ejection fraction of the heart may decrease to the point that the patient develops cardiogenic shock. This is difficult to treat. Other causes of cardiogenic shock include the 'mechanical' complications such as left ventricular free wall rupture as listed below. Patients may require inotropic support and/or an intra-aortic balloon pump.
As described above, if the patient survives the acute phase their ventricular myocardium may be dysfunctional resulting in chronic heart failure. Loop diuretics such as furosemide will decrease fluid overload. Both ACE-inhibitors and beta-blockers have been shown to improve the long-term prognosis of patients with chronic heart failure.
Ventricular fibrillation, as mentioned above, is the most common cause of death following a MI. Other common arrhythmias including ventricular tachycardia.
Atrioventricular block is more common following inferior myocardial infarctions.
Pericarditis in the first 48-72 hours following a transmural MI is common (c. 10% of patients). It is due to a reaction to the transmural necrosis.
The inflammation affects the adjacent visceral and parietal pericardium; in other words, the inflammation is usually localized to the region of the pericardium overlying the necrotic myocardial segment. The pain is typical for pericarditis (worse on lying flat etc), a pericardial rub may be heard and a pericardial effusion may be demonstrated with an echocardiogram.